Search results

John P. Robinson, University of Maryland; Jonathan Gershuny, University of Oxford; 2012-006

Increasing number of studies have investigated the impact of maternal exposure to air pollution during pregnancy and adverse birth outcomes, particularly low birth weight (LBW, <2,500 g at birth) and preterm birth (PTB, <37 completed weeks of gestation). We performed a comprehensive review of the peer-reviewed literature and a meta-analysis to quantify the association between maternal exposure to particulate matter with aerodynamic diameter 2.5 and 10 μm (PM 2.5  and PM 10 ) during pregnancy and the risk of LBW and PTB. We identified 20 peer-reviewed articles providing quantitative estimate of exposure and outcome that met our selection criteria. There was significant heterogeneity between studies, particularly for findings related to PM 10  exposure (LBW,  I -squared 54%,  p  = 0.01; PTB,  I -squared = 73%,  p  < 0.01). Results from random-effect meta-analysis suggested a 9% increase in risk of LBW associated with a 10-μg/m 3  increase in PM 2.5  (combined odds ratios (OR), 1.09; 95% confidence interval (CI), 0.90–1.32), but our 95% CI included the null value. We estimated a 15% increase in risk of PTB for each 10-μg/m 3  increase in PM 2.5  (combined OR, 1.15; CI, 1.14–1.16). The magnitude of risk associated with PM 10  exposure was smaller (2% per 10-μg/m 3  increase) and similar in size for both LBW and PTB, neither reaching formal statistical significance. We observed no significant publication bias, with  p  > 0.05 based on both Begg's and Egger's bias tests. Our results suggest that maternal exposure to PM, particularly PM 2.5  may have adverse effect on birth outcomes. Additional mechanistic studies are needed to understand the underlying mechanisms for this association.

John P. Robinson, University of Maryland, et al.; 2012-005

John P. Robinson, University of Maryland; William Rivers, Integrated Technical Solutions; Paul Harwood, University of North Florida; 2012-004

The purpose of this article is to examine the relationship between environmental changes and infectious diseases and their impact on health in environmental justice (EJ) communities. The evolution of EJ science and research is contingent upon an integrated approach that takes into account social processes and environmental changes to address the burden of infectious diseases in EJ communities. We recognize that infectious disease and environmental justice is novel and calls for more research in this area, especially as the focus of public health shifts towards an ecologic and social approach to disease prevention. We attempt to explore in further detail how environmental changes such as urbanization, agriculture, and climate variability could potentially influence pathogen dynamics, vector transmission, host susceptibility, and disease outcomes among environmental justice populations.

Faculty Associate Kevin Roy co-authors book on public policy impacts on fatherhood

Faculty Associate John Haltiwanger's paper with two Census Bureau economists challenges conventional wisdom

Faculty Associate Melissa Kearney and Philip B. Levine find that inequality trumps location in predicting early childbearing out of wedlock

Faculty Associate Feinian Chen studies health implications for grandparents caring for grandchildren

Faculty Associate David Segal oversees research into the impact of social change on the military